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AstraZeneca ltd computer system pclab
Computer System Pclab, supplied by AstraZeneca ltd, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/computer+system+pclab/computer+system+pclab/pm21186275-99-12-15
Average 90 stars, based on 1 article reviews
computer system pclab - by Bioz Stars, 2026-10
90/100 stars

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Article Title: Increased expression of monocarboxylate transporter 1 after acute ischemia of isolated, perfused mouse hearts.
Article Snippet: Contents lists available at ScienceDirect Life Sciences j ourna l homepage: www.e lsev ie r.com/ locate / l i fesc ie Increased expression of monocarboxylate transporter 1 after acute ischemia of isolated, perfused mouse hearts Vladimir Martinov a,1, Syed Mohammad Husain Rizvi a,1, Stian Andre Weiseth a, Julia Sagave a, Linda H. Bergersen b,c, Guro Valen a,⁎ a Department of Physiology, Institute of Basic Medical Sciences, University of Oslo, Oslo, Norway b Centre for Molecular Biology and Neuroscience, Institute of Basic Medical Sciences, University of Oslo, Oslo, Norway c Department of Anatomy, Institute of Basic Medical Sciences, University of Oslo, Oslo, Norway ⁎ Corresponding author.. Institute of Basic Medical Scie Postbox 1103 Blindern, 0317 Oslo, Norway.. Tel.

Article Title: Mechanisms of novel cardioprotective functions of CCN2/CTGF in myocardial ischemia-reperfusion injury.
Article Snippet: M. Shakil Ahmed, Jørgen Gravning, Vladimir N. Martinov, Thomas G. von Lueder, Thor Edvardsen, Gabor Czibik, Ingvild T. Moe, Leif E. Vinge, Erik Øie, Guro Valen, and Håvard Attramadal Institute for Surgical Research, Department of Cardiology, and Center for Heart Failure Research, Oslo University Hospital-Rikshospitalet and University of Oslo and Department of Physiology, Institute of Basic Medical Sciences, and Center for Heart Failure Research, University of Oslo, Oslo, Norway

Article Title: Myocardial protection evoked by hyperoxic exposure involves signaling through nitric oxide and mitogen activated protein kinases.
Article Snippet: J. Vaage Ullevål University Hospital Oslo, Norway ■ Abstract Background Hyperoxic exposure in vivo (> 95 % oxygen) attenuates ischemia-reperfusion injury, but the signaling mechanisms of this cardioprotection are not fully determined.. We studied a possible role of nitric oxide (NO) and mitogen activated protein kinases (MAPK) in hyperoxic protection.. Methods Mice (n = 7–9 in each group) were kept in normoxic or hyperoxic environments for 15 min prior to harvesting the heart and Langendorff perfusion with global ischemia (45 min) and reperfusion (60 min).



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